Anti-Human CD64 (Fc gamma Receptor 1) FITC

Also known as: FcgammaR1, Fcgamma R1, FcgR1

Clone: 10.1

RUO: For Research Use Only. Not for use in diagnostic procedures.

SKU# 11-0649

Cat. No. Size
11-0649-41 25 tests
11-0649-42 100 tests
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Data for Anti-Human CD64 (Fc gamma Receptor 1) FITC.

Staining of normal human peripheral blood cells with Mouse IgG1 kappa Isotype Control FITC (cat....View More

  • Data for Anti-Human CD64 (Fc gamma Receptor 1) FITC.
Description

Description: The 10.1 monoclonal antibody reacts with human CD64 (FcRI), a 75 kDa type I transmembrane protein. CD64 is the high affinity receptor for IgG and is involved in antibody-dependent cell-mediated cytotoxicity (ADCC), phagocytosis, and regulation of cytokine production. Monocytes and macrophages express CD64, while mature granulocytes and lymphocytes are negative.

Details
Host Mouse
Isotype IgG1, kappa
Reactivity Human
Conjugate FITC
Laser Blue Laser
Emit 518 nm
Excite 488 nm
Reported Applications Flow Cytometric Analysis
Documentation
TDS Link Download TDS
Additional Formats
Cat. No. Name Excite Emit Application Reg.
16-0649 Anti-Human CD64 (Fc gamma Receptor 1) Functional Grade Purified FC, IHC RUO
12-0649 Anti-Human CD64 (Fc gamma Receptor 1) PE 488 - 561 nm FC RUO
17-0649 Anti-Human CD64 (Fc gamma Receptor 1) APC 633 - 647 nm 660 nm FC RUO
14-0649 Anti-Human CD64 (Fc gamma Receptor 1) Purified FC, IHC RUO
46-0649 Anti-Human CD64 (Fc gamma Receptor 1) PerCP-eFluor® 710 488 nm 710 nm FC RUO
Related Products
Cat. No. Name Excite Emit Application Reg.
11-4714 Mouse IgG1 K Isotype Control FITC 488 nm FC, ICC, IHC RUO
References

References: Kishimoto, T., A.E.G., von dem Borne, et al. eds. (1998). Leucocyte Typing VI: White Cell Differentiation Antigens. Garland Publishing Inc. London.

Schlossman, S., L. Bloumsell, et al. eds (1995). Leucocyte Typing V: White Cell Differentiation Antigens. Oxford University Press. New York.

McMichael, A.J., P.C.L. Beverly, et al. eds. (1987). Leucocyte Typing III: White Cell Differentiation Antigens. Oxford University Press. New York.


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