Anti-Human CD25 APC

Also known as: Interleukin-2 Receptor alpha, IL-2Ra, IL2Ra, Tac

Clone: BC96

RUO: For Research Use Only. Not for use in diagnostic procedures.

SKU# 17-0259

Cat. No. Size
17-0259-41 25 tests
17-0259-42 100 tests
Please inquire about custom configurations or bulk packaging.

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Data for Anti-Human CD25 APC.

Staining of normal human peripheral blood cells with Anti-Human CD4 PE (cat. 12-0049) and Mouse IgG1...View More

  • Data for Anti-Human CD25 APC.
Description

Description: The BC96 monoclonal antibody reacts with human CD25, the 55 kDa interleukin-2 receptor α chain (IL-2Rα). CD25 is expressed by early progenitors of T and B lineage as well as by activated mature T and B lymphocytes. By itself, CD25 binds IL-2 only with low affinity. However, CD25 associates with CD122 (IL-2 receptor β chain) and CD132 (common γ chain) to form the high affinity IL-2 receptor. CD25 plays a role in lymphocyte differentiating and activation/proliferation.

Details
Host Mouse
Isotype IgG1, kappa
Reactivity Human
Conjugate APC
Laser Red Laser
Emit 660 nm
Excite 633 - 647 nm
Reported Applications Flow Cytometric Analysis
Documentation
TDS Link Download TDS
Additional Formats
Cat. No. Name Excite Emit Application Reg.
45-0259 Anti-Human CD25 PerCP-Cyanine5.5 488 nm FC RUO
13-0259 Anti-Human CD25 Biotin FC RUO
53-0259 Anti-Human CD25 Alexa Fluor® 488 488 nm 519 nm FC RUO
48-0259 Anti-Human CD25 eFluor® 450 405 nm 450 nm FC RUO
25-0259 Anti-Human CD25 PE-Cyanine7 488 - 561 nm 775 nm FC RUO
47-0259 Anti-Human CD25 APC-eFluor® 780 633 - 647 nm 780 nm FC RUO
14-0259 Anti-Human CD25 Purified FC RUO
15-0259 Anti-Human CD25 PE-Cyanine5 488 - 561 nm FC RUO
12-0259 Anti-Human CD25 PE 488 - 561 nm 575 nm FC RUO
Related Products
Cat. No. Name Excite Emit Application Reg.
12-0049 Anti-Human CD4 PE 488 - 561 nm 575 nm FC RUO
17-4714 Mouse IgG1 K Isotype Control APC 633 - 647 nm 660 nm FC RUO
References

References:
Schlossman, S., L. Bloumsell, et al. eds. (1995). Leucocyte Typing V: White Cell Differentiation Antigens. Oxford University Press. New York.